Good afternoon,
Given the state of modern cycling at the end of the 2024 season with many more riders jumping into LT1 / Z2 steady state training , it’s fairly safe to say that in the senior categories most if not all riders have done some kind of targeted z2 training following loosely or closely to the ideas being shared around the internet.
This thread concerns the future as athletes look to leverage their z2 gains and become the most competitive build possible for the sport.
So begs the question, with highly trained endurance athletes who are completing 1000TSS weeks 5h z2 rides w/ mixed sprints, 3-4h z1/z2 rides etc week after week, is there a plateau effect waiting up there wherein the athletes have become so oxidatively powerful that their base levels of muscle mass do not drive sufficient levels of lactate production to take full advantage of this large oxidative capacity.
The idea of needing to raise VLAmax as the season turns from base to build is already documented, this however would be a more novel approach, understanding that VLAmax on a per muscle fiber basis can only be so high, the only way to sufficiently raise VLAmax to be in balance with monstrous base training would be by gaining muscle mass.
In other words, could a highly trained athlete raise their effective V02max by increasing their muscle mass , and is this the effect that we are seeing with Jonas Abrahamsen who allegedly gained almost 20kg and was then in contention for the KOM jersey of the tour.
The idea being that without the extra muscles in the legs he was not able to deliver sufficient load to his highly trained base systems and as a result had a lower effective vo2max unadjusted